Aids Using Information To Combat Spreading

3608 words - 15 pages

Intro Sample...

AIDS - What's new? Is the message getting through? We already know enough about AIDS to prevent its spread, but ignorance, complacency, fear and bigotry continue to stop many from taking adequate precautions. We know enough about how the infection is transmitted to protect ourselves from it without resorting to such extremes as mandatory testing, enforced quarantine or total celibacy. But too few people are heeding the AIDS message. Perhaps many simply don't like or want to believe what they hear, preferring to think that AIDS "can't happen to them." Experts repeatedly remind us that infective agents do not discriminate, but can infect any and everyone. Like other communicable diseases, AIDS can strike anyone. It is not necessarily... View More »

Body Sample...

Thus HIV may not produce illness until its genes are "turned on" five, ten, fifteen or perhaps more years after the initial infection. During the latent period, HIV carriers who harbour the virus without any sign of illness can unknowingly infect others. On average, the dormant virus seems to be triggered into action three to six years after first invading human cells. When switched on, viral replication may speed along, producing new viruses that destroy fresh lymphocytes. As viral replication spreads, the lymphocyte destruction virtually sabotages the entire immune system. In essence, HIV viruses do not kill people, they merely render the immune system defenceless against other "opportunistic: infections, e.g. yeast invasions, toxoplasmosis, cytomegalovirus and Epstein Barr infections, massive herpes infections, special forms of pneumonia (Pneumocystis carinii - the killer in half of all AIDS patients), and otherwise rare malignant tumours (such as Kaposi's sarcoma.) Cofactors may play a crucial contributory role:

What prompts the dormant viral genes suddenly to burst into action and start destroying the immune system is one os the central unsolved challenges about AIDS. Some scientists speculate that HIV replication may be set off by cofactors or transactivators that stimulate or disturb the immune system. Such triggers may be genetically determined proteins in someone's system, or foreign substances from other infecting organisms - such as syphilis, chlamydia, gonorrhea, HTLV-1 (leukemia), herpes, or CMV (cytomegalovirus) - which somehow awaken the HIV virus. ...

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